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Arl2 (NM_019722) Mouse Tagged ORF Clone Lentiviral Particle
CAT＃: MR201694L3V
- LentiORF®
-
Lenti ORF particles, Arl2 (Myc-DDK-tagged) - Mouse ADP-ribosylation factor-like 2 (Arl2), 200ul, >10^7 TU/mL
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CNY 7,410.00
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Specifications
| Product Data | |
| Product Name | Arl2 (NM_019722) Mouse Tagged ORF Clone Lentiviral Particle |
| Synonyms | 2610009M23Rik; AI115441; AW553335 |
| Vector | pLenti-C-Myc-DDK-P2A-Puro |
| ACCN | NM_019722 |
| ORF Size | 555 bp |
| Sequence Data |
The ORF insert of this clone is exactly the same as(MR201694).
|
| OTI Disclaimer | The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info |
| OTI Annotation | This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene. |
| Reference Data | |
| RefSeq | NM_019722.3, NP_062696.2 |
| RefSeq Size | 869 bp |
| RefSeq ORF | 555 bp |
| Locus ID | 56327 |
| Gene Summary | Small GTP-binding protein which cycles between an inactive GDP-bound and an active GTP-bound form, and the rate of cycling is regulated by guanine nucleotide exchange factors (GEF) and GTPase-activating proteins (GAP). GTP-binding protein that does not act as an allosteric activator of the cholera toxin catalytic subunit. Regulates formation of new microtubules and centrosome integrity. Prevents the TBCD-induced microtubule destruction. Participates in association with TBCD, in the disassembly of the apical junction complexes. Antagonizes the effect of TBCD on epithelial cell detachment and tight and adherens junctions disassembly. Together with ARL2, plays a role in the nuclear translocation, retention and transcriptional activity of STAT3. Component of a regulated secretory pathway involved in Ca(2+)-dependent release of acetylcholine. Required for normal progress through the cell cycle.[UniProtKB/Swiss-Prot Function] |
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