PEX3 (NM_003630) Human Tagged ORF Clone Lentiviral Particle

CAT#: RC202031L2V

  • LentiORF®

Lenti ORF particles, PEX3 (mGFP-tagged) - Human peroxisomal biogenesis factor 3 (PEX3), 200ul, >10^7 TU/mL

ORF Plasmid: DDK tGFP



Need custom lentivirus service?
Get a free quote

CNY 9,975.00


货期*
详询

规格
    • 200 ul

Product images

经常一起买 (3)
Lenti ORF control particles of pLenti-C-mGFP, >10^7 TU/mL, 0.5 mL
    • 500 ul

CNY 2,950.00


One-Wash Lentivirus Titer Kit, HIV-1 p24 ELISA
    • 96 reactions

CNY 5,230.00


Rabbit Polyclonal Anti-PEX3 Antibody
    • 100 ug

CNY 5,250.00

Specifications

Product Data
Product Name PEX3 (NM_003630) Human Tagged ORF Clone Lentiviral Particle
Synonyms PBD10A; PBD10B; TRG18
Vector pLenti-C-mGFP
ACCN NM_003630
ORF Size 1119 bp
Sequence Data
The ORF insert of this clone is exactly the same as(RC202031).
OTI Disclaimer The molecular sequence of this clone aligns with the gene accession number as a point of reference only. However, individual transcript sequences of the same gene can differ through naturally occurring variations (e.g. polymorphisms), each with its own valid existence. This clone is substantially in agreement with the reference, but a complete review of all prevailing variants is recommended prior to use. More info
OTI Annotation This clone was engineered to express the complete ORF with an expression tag. Expression varies depending on the nature of the gene.
Reference Data
RefSeq NM_003630.1
RefSeq Size 2774 bp
RefSeq ORF 1122 bp
Locus ID 8504
Domains Peroxin-3
Protein Families Druggable Genome
MW 42.1 kDa
Gene Summary The product of this gene is involved in peroxisome biosynthesis and integrity. It assembles membrane vesicles before the matrix proteins are translocated. Peroxins (PEXs) are proteins that are essential for the assembly of functional peroxisomes. The peroxisome biogenesis disorders (PBDs) are a group of genetically heterogeneous autosomal recessive, lethal diseases characterized by multiple defects in peroxisome function. The peroxisomal biogenesis disorders are a heterogeneous group with at least 14 complementation groups and with more than 1 phenotype being observed in cases falling into particular complementation groups. Although the clinical features of PBD patients vary, cells from all PBD patients exhibit a defect in the import of one or more classes of peroxisomal matrix proteins into the organelle. Defects in this gene are a cause Zellweger syndrome (ZWS). [provided by RefSeq, Oct 2008]
*Delivery time may vary from web posted schedule. Occasional delays may occur due to unforeseen complexities in the preparation of your product. International customers may expect an additional 1-2 weeks in shipping.
Customer Reviews 
Loading...